Mitochondrial Communications

Open access

ISSN: 2590-2792

Mitochondrial Communications

Open access

Mitochondrial Communications is committed to the rapid publication of high quality, rigorously peer reviewed research articles, reviews, commentaries on scientifically sound and clinically important i...

Mitochondrial Communications is committed to the rapid publication of high quality, rigorously peer reviewed research articles, reviews, commentaries on scientifically sound and clinically important issues and letters to editor. The journal covers a wide range of topics in basic research of mitochondrial biology and the clinical aspects of mitochondrial diseases.

Subjects considered for publication include, but are not limited to:

  • all aspects of mitochondrial biology, including mitochondrial dynamics and quality control, mitochondria-organelle interactions, mitochondrial genetics and evolution, mitochondria-nuclear crosstalk, mitochondrial physiology and stress responses
  • Redox and oxidative stress
  • stem cell
  • cell death and differentiation
  • cell signaling
  • immunity
  • iron metabolism
  • cell metabolism
  • aging and aging related diseases

The journal provides a platform for basic and clinical researchers to discuss the exciting and dynamic fields of mitochondrial research. Thus, clinical features of mitochondrial diseases such as (but not limited to) cancer, metabolic syndromes, myopathies, neurodegenerative diseases and heart diseases are among the major topics of the journal.

Mitochondrial Communications is a high quality, fully Open Access journal: all accepted articles will be immediately and permanently available on ScienceDirect, free to read, download and share.

Mitochondria: The hot organelles are getting hotter

OPA1-Exon4b modulates the migration and invasion of hepatocellular carcinoma cells by ATP regulation

The metabolism and function of phospholipids in Mitochondria

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1. The metabolism and function of phospholipids in Mitochondria

2. Correcting abnormal mitochondrial dynamics to facilitate tumor treatment

3. Structure-destabilizing mutations unleash an intrinsic perforation activity of antiapoptotic Bcl-2 in the mitochondrial membrane enabling apoptotic cell death

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News

KeAi Talks: Mitochondrial Communications serial webinar 2

Online event: Mitochondria as Qi revisited: A mitochondrial etiology of common diseases

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Call for papers

Special issue on the protocols and methods of mitochondrial research

Submissions deadline: 31 March 2024

Special issue on mitochondrial DNA and cancer

Submissions close: 31 December 2023

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